Just as Radio Frequencies can correct Radio frequency fields, transthyretin, and Alzheimer'sdisease The Same is to the Cause of Alzheimer by cell piracy

Radiofrequencyfield (RF) exposure provided cognitive benefits in an
animal study. InAlzheimer'sdisease (AD)
www.ncbi.nlm.nih.gov/pubmed/20164553
Radiofrequency field (RF) exposure provided cognitive benefits in an
animal study. In Alzheimer's disease(AD) mice, exposure reduced brain
amyloid-β (Aβ) deposition through decreased aggregation of Aβ and
increase in soluble Aβ levels. Basedon our studies on humans on RF
from wireless phones, we propose that transthyretin (TTR) might
explain the findings. In a cross-sectional study on 313 subjects, we
used serum TTR as a marker of cerebrospinal fluid TTR. We found a
statistically significantly positive βcoefficient for TTR for time
since first use of mobile phones and desktop cordless phones combined
(P=0.03). The electromagnetic fieldparameters were similar for the
phone types. In a provocation study on 41 persons exposed for 30 min
to an 890-MHz GSM signal with specific absorption rate of 1.0 Watt/kg
to the temporal area of thebrain, we found statistically significantly
increased serum TTR 60 min after exposure. In our cross-sectional
study, use of oral snuff also yielded statistically significantly
increased serum TTR concentrations and nicotine has been associated
with decreased riskfor AD and to upregulate the TTR gene in choroid
plexus but not in the liver, another source of serum TTR. TTR
sequesters Aβ, thereby preventing the formation of Aβ plaques in the
brain. Studies have shown that patients with AD have lowered TTR
concentrations in the cerebrospinal fluid and have attributed the
onset of AD to insufficient sequestering of Aβ by TTR. We propose that
TTR might be involved in the findings of RF exposure benefit in AD
mice.
Keywords
Blood-cerebrospinal barrier, cordless phone, DECT, dementia, mobile
phone, nicotine, plexus choroideus

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